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Questions, however, remain about potential adverse effects, as, paradoxically, the transient presence of senescent cells could be beneficial during embryogenesis and wound healing.82 A more conservative approach involves the use of molecules with senomorphic effects capable of modulating the counterproductive nature of senescent cells and restarting cell proliferation

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Am J Alzheimers Dis Other Demen (2020) 35:1533317519899546

Nicotinamide adenine dinucleotide (NADH) and acetyl-CoA, two metabolic intermediates produced in the course of FAO, induce activation of PDK, which in turn phospho-inactivates the E1 subunit of the PDH complex, leading to lower rates of glucose oxidation and higher rates of lactate release 70